Here, we investigated the suppressive activity of microglia during experimental autoimmune encephalomyelitis (EAE) induced by myelin oligodendrocyte glycoprotein, with the goal of understanding their part in regulating the T cell reaction. cells nitric oxide (NO), the production of which was
Our results claim that PD-1/CTLA-4 blockade escalates the capability of T cells to infiltrate EBV-induced lymphomas, also to become highly activated (while measured by NFATC1 nuclear translocation)
Our results claim that PD-1/CTLA-4 blockade escalates the capability of T cells to infiltrate EBV-induced lymphomas, also to become highly activated (while measured by NFATC1 nuclear translocation). EBV-induced lymphomas in wire blood-humanized mice when began 10 times after injection of